| Ditemukan 569 dokumen dengan kata kunci 8781 |
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Retrospective cohort study of familial hypomagnesaemia with hypercalciuria and nephrocalcinosis due to CLDN16 mutations
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 30, No.4, April 2015: p.636–644 |
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Koleksi: Artikel Internasional - Oxford ::
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Inherited disorders of renal hypomagnesaemia
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 29, suppl 4, September 2014: p.iv63–iv71 |
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Koleksi: Artikel Internasional - Oxford ::
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Chronic hypercalcaemia from inactivating mutations of vitamin D 24-hydroxylase (CYP24A1): implications for mineral metabolism changes in chronic renal failure
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 29, No.3, March 2014: p.636–643 |
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Koleksi: Artikel Internasional - Oxford ::
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Microscopic nephrocalcinosis in chronic kidney disease patients
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 30, No.5, May 2015: p.843–848 |
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Koleksi: Artikel Internasional - Oxford ::
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Cisplatin-induced injury of the renal distal convoluted tubule is associated with hypomagnesaemia in mice
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 28, No.4, April 2013: p.879–889 |
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Koleksi: Artikel Internasional - Oxford ::
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Toxicity and clinical outcomes in patients with HIV on zidovudine and tenofovir based regimens: a retrospective cohort study
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| No. Panggil: Transactions of The Royal Society of Tropical Medicine and Hygiene;Volume 109 No.6 Juni 2015 p 379–385 |
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Koleksi: Artikel Internasional - Oxford ::
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Pre-dialysis hospital use and late referrals in incident dialysis patients in England: a retrospective cohort study
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 30, No.1, January 2015: p.124–129 |
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Koleksi: Artikel Internasional - Oxford ::
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Familial C3 glomerulonephritis associated with mutations in the gene for complement factor B
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| No. Panggil: Neophorlogy Dialysis Transplantation; Volume 30, No.5, May 2015: p.862–864 |
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Koleksi: Artikel Internasional - Oxford ::
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A nationwide survey of AicardiGoutie` res syndrome patients identifies a strong association between dominant TREX1 mutations and chilblain lesions: Japanese cohort study
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Familial dilated cardiomyopathy mutations uncouple troponin I phosphorylation from changes in myofibrillar Ca21 sensitivity
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